Orthopedic Problems in Seniors

November 08, 2024
Orthopädische Probleme bei Senioren
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A comprehensive overview of diseases, nutrition and lifestyle.

Demographic change is leading to a steadily growing number of older people in society. However, with age, the risks of various orthopedic diseases also increase, which can significantly affect the quality of life and independence of seniors. The most common orthopedic problems include osteoporosis, osteoarthritis and sarcopenia. This article takes a detailed look at these diseases, possible challenges and how targeted nutrition and lifestyle changes can help prevent and improve them.  

Common orthopedic diseases in seniors  

Osteoporosis and its effects on bone health

Osteoporosis is characterized by reduced bone mass and deterioration of bone tissue structure, leading to increased fragility and an increased risk of fractures. The decrease in bone density is often the result of an imbalance between bone breakdown and formation, which is accelerated by hormonal changes such as estrogen deficiency after menopause in women. Men are also affected, but to a lesser extent and often at an older age. 

Several risk factors contribute to the development of osteoporosis, including genetic predisposition, low BMI (body mass index), smoking, excessive alcohol consumption and certain medications such as glucocorticoids. The disease often remains asymptomatic until a fracture occurs, which underlines the importance of preventive measures.  

Influence of nutrients:  

  1. Protein: Adequate protein intake (1.0–1.2 g/kg body weight per day) is crucial for bone health. Protein supports collagen synthesis, the structure of bone, and increases insulin-like growth factor 1 (IGF-1), which promotes bone formation.
  2. Calcium : Essential for bone mineralization; a daily intake of 1,000–1,200 mg is recommended. Good sources include dairy products, green leafy vegetables and fortified foods.
  3. Vitamin D: Promotes calcium absorption in the intestine. A deficiency can lead to secondary hyperparathyroidism, which accelerates bone loss. An intake of 800–2,000 IU per day is often necessary.
  4. Vitamin K : Important for the carboxylation of osteocalcin, a protein that supports bone mineralization. Sources include green leafy vegetables such as spinach and kale.
  5. Magnesium : Involved in bone structure and calcium metabolism. A deficiency can impair bone strength. Found in whole grains, nuts and seeds.
  6. Zinc : Supports osteoblast activity and bone formation. Sources include meat, seafood, legumes and nuts.
  7. Copper and manganese: Involved in collagen cross-linking and bone formation. Found in nuts, seeds and whole grains.
  8. Vitamin C : Important for collagen synthesis and antioxidant defense. Abundant in citrus fruits, berries and peppers.
  9. B vitamins (B6, B12, folic acid): High homocysteine ​​concentrations caused by deficiencies in these vitamins are associated with an increased risk of fractures.

 

Osteoarthritis and its influences on joint function

Osteoarthritis is a degenerative joint disease characterized by the breakdown of articular cartilage and changes in the subchondral bone. This leads to pain, stiffness and limited mobility of the affected joints. The pathophysiology of osteoarthritis includes both mechanical factors such as excessive strain on the joints and biochemical processes that lead to inflammation and cartilage degeneration.  

Risk factors for osteoarthritis include advanced age, obesity, previous joint injuries, genetic predisposition and certain occupations that require repetitive movements or strain. Being overweight not only increases the mechanical load on the joints, but also promotes the production of pro-inflammatory cytokines in the fatty tissue, which can accelerate the disease process.  

Influence of nutrients:  

  1. Healthy fats: Reducing saturated fats and trans fats in favor of monounsaturated and polyunsaturated fatty acids can reduce inflammation.
  2. Omega-3 fatty acids: Have anti-inflammatory properties and can relieve symptoms. Sources include fatty fish (salmon, mackerel), flax seeds and walnuts.
  3. Antioxidants ( vitamins C and E): Reduce oxidative stress in the joints. Abundant in fruits, vegetables, nuts and seeds.
  4. Vitamin D: May have anti-inflammatory effects and improve joint function.
  5. Selenium: Antioxidant trace element that can reduce inflammation. Sources include Brazil nuts, fish and grains.
  6. Flavonoids and polyphenols: Plant compounds with antioxidant and anti-inflammatory properties, found in berries, grapes, green tea and olive oil.
  7. Glucosamine and chondroitin: substances produced by the body that support cartilage metabolism. Supplementation can improve symptoms in some patients.

 

Sarcopenia and age-related muscle loss

Sarcopenia is the age-related loss of muscle mass and function, which leads to muscle weakness, increased susceptibility to falls and reduced quality of life. The pathophysiology of sarcopenia is multifactorial and includes hormonal changes, chronic inflammation, oxidative stress, reduced nerve supply to the muscles and reduced protein synthesis.  

Risk factors for the development of sarcopenia include physical inactivity, inadequate protein intake, chronic diseases such as diabetes and heart failure, and social factors such as isolation and poverty. Adequate protein intake is crucial to slow muscle breakdown and promote muscle protein synthesis. In particular, the amino acid leucine plays an important role in stimulating muscle protein synthesis.  

Influence of nutrients:  

  1. Protein: An increased protein intake (1.2-1.5 g/kg body weight per day) is necessary to stimulate muscle protein synthesis. Branched-chain amino acids such as leucine are particularly important.
  2. Carbohydrates: Necessary for energy supply and support protein utilization.
  3. Vitamin D : Supports muscle strength and balance, deficiency is associated with muscle weakness.
  4. Antioxidants: Protection against oxidative stress, which promotes muscle breakdown.
  5. Vitamin B12 and folic acid: Important for nerve function and muscle coordination.
  6. Creatine: May increase muscle strength and mass, supplementation may be beneficial in older adults.

 

The importance of regular physical activity in old age  

In addition to diet, regular physical activity is a crucial factor in maintaining bone and muscle health. Strength training can increase muscle mass and strength, improve bone density and reduce the risk of falls. Weight-bearing exercises such as walking, dancing or light jogging stimulate bone metabolism and promote bone formation.  

Balance and flexibility exercises such as Tai Chi or yoga can improve coordination and help prevent falls. A combination of endurance, strength and balance exercises is recommended for older adults to maintain physical function and quality of life. Activities should be adapted to individual abilities and health conditions, ideally under the supervision of a professional.  

 

Challenges and solutions for implementation  

Despite the known benefits, seniors may have difficulty implementing diet and exercise recommendations. Loss of appetite, difficulty chewing and swallowing, sensory changes such as taste and smell disturbances, social isolation and financial limitations can affect food intake. Medications can affect nutrient absorption or cause side effects such as nausea.  

Individual counseling from nutrition professionals can help identify barriers and develop tailored solutions. For example, energy-dense and high-protein snacks can be introduced, or the consistency of food can be adjusted to make eating easier. Community programs and social support can increase motivation and improve compliance.  

A multidisciplinary approach involving physicians, nutritionists, physical therapists and other health professionals is critical to success. Regular monitoring and adjustment of therapy can help maximize the effectiveness of interventions and minimize potential side effects or interactions.  

 

Sources:  

  1. World Health Organization. Global Report on Falls Prevention in Older Age . 2007, www.whogis.com .  
  2. Minetto, Marco A., et al. "Common Musculoskeletal Disorders in the Elderly: The Star Triad." Journal of Clinical Medicine , vol. 9, no. 4, 2020, p. 1216, doi:10.3390/jcm9041216.  
  3. Le, Ngoc Hoan, et al. "Quercetin Protects Against Obesity-Induced Skeletal Muscle Inflammation and Atrophy." Mediators of Inflammation , vol. 2014, Article ID 834294, 2014, doi:10.1155/2014/834294.  
  4. Fioravanti, Alberto, and Gioconda Collodel. "In Vitro Effects of Chondroitin Sulfate." Advances in Pharmacology , vol. 53, 2006, pp. 449-465, doi:10.1016/S1054-3589(05)53022-9.  
  5. Cruz-Jentoft, Alfonso J., and Avan A. Sayer. "Sarcopenia." The Lancet , vol. 393, no. 10191, 2019, pp. 2636-2646, doi:10.1016/S0140-6736(19)31138-9.  
  6. Valdes, Ana M., and Joanne Stocks. "Osteoarthritis and Aging." EMJ , vol. 3, no. 1, 2018, pp. 116-123.  
  7. Arden, Nigel, et al. Atlas of Osteoarthritis . Springer Healthcare Limited, 2014.  
  8. Altman, R., et al. "The American College of Rheumatology Criteria for the Classification and Reporting of Osteoarthritis of the Hip." Arthritis and Rheumatism , vol. 34, no. 5, 1991, pp. 505-514, doi:10.1002/art.1780340502.  
  9. Bruyère, Olivier, et al. "An Updated Algorithm Recommendation for the Management of Knee Osteoarthritis from the European Society for Clinical and Economic Aspects of Osteoporosis, Osteoarthritis and Musculoskeletal Diseases (ESCEO)." Seminars in Arthritis and Rheumatism , vol. 49, no. 3, 2019, pp. 337-350, doi:10.1016/j.semarthrit.2019.04.008.  
  10. Hochberg, Marc C., et al. "American College of Rheumatology 2012 Recommendations for the Use of Nonpharmacologic and Pharmacologic Therapies in Osteoarthritis of the Hand, Hip, and Knee." Arthritis Care & Research , vol. 64, no. 4, 2012, pp. 465-474, doi:10.1002/acr.21596.  
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  13. Bae, Seong-Hwan, et al. "Position Statement: Exercise Guidelines for Osteoporosis Management and Fall Prevention in Osteoporosis Patients." Journal of Bone Metabolism , vol. 30, no. 2, 2023, pp. 149-165, doi:10.11005/jbm.2023.30.2.149.  
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